« Previous
Next »
Journal of Molecular and Cellular Cardiology
Volume 46, Issue 3
, Pages 385-394
, March 2009
Distinct contractile and molecular differences between two goat models of atrial dysfunction: AV block-induced atrial dilatation and atrial fibrillation
References
- . Stroke prevention. An opportunity for efficient utilization of health care resources during the coming decade. Stroke. 1994;25:220–224
- . Ischemic stroke subtypes: a population-based study of incidence and risk factors. Stroke. 1999;30:2513–2516
- . Left atrial size and the risk of ischemic stroke in an ethnically mixed population. Stroke. 1999;30:2019–2024
- . Risk for systemic embolization of atrial fibrillation without mitral stenosis. Am. J. Cardiol. 1990;65:1112–1116
- . Left atrial size and the risk of stroke and death. The Framingham Heart Study. Circulation. 1995;92:835–841
- . Remodeling of atrial dimensions and emptying function in canine models of atrial fibrillation. Cardiovasc. Res. 2001;52:217–225
- . Mechanical remodeling of the left atrium after loss of atrioventricular synchrony — a long-term study in humans. Circulation. 1999;100:1714–1721
- . Electrical and contractile remodeling during the first days of atrial fibrillation go hand in hand. Circulation. 2003;107:1433–1439
- Atrial fibrillation-induced atrial contractile dysfunction: a tachycardiomyopathy of a different sort. Cardiovasc. Res. 2002;53:192–201
- Impaired myofibrillar energetics and oxidative injury during human atrial fibrillation. Circulation. 2001;104:174–180
- . Remodeling of Ca2+-handling by atrial tachycardia: evidence for a role in loss of rate-adaptation. Cardiovasc. Res. 2002;54:416–426
- Cellular mechanisms of depressed atrial contractility in patients with chronic atrial fibrillation. Circulation. 2001;103:691–698
- Moderate and chronic hemodynamic overload of sheep atria induces reversible cellular electrophysiologic abnormalities and atrial vulnerability. J. Am. Coll. Cardiol. 2004;44:1918–1926
- Development of a substrate of atrial fibrillation during chronic atrioventricular block in the goat. Circulation. 2005;111:30–37
- Direction-dependent conduction abnormalities in a canine model of atrial fibrillation due to chronic atrial dilatation. Am. J. Physiol. Heart Circ. Physiol. 2004;287:H634–644
- AVE0118, blocker of the transient outward current (I(to)) and ultrarapid delayed rectifier current (I(Kur)), fully restores atrial contractility after cardioversion of atrial fibrillation in the goat. Circulation. 2006;114:1234–1242
- . Structural changes of atrial myocardium due to sustained atrial fibrillation in the goat. Circulation. 1997;96:3157–3163
- . Rapid cooling contractures as an index of sarcoplasmic reticulum calcium content in rabbit ventricular myocytes. Am. J. Physiol. 1989;257:H1369–1377
- . Reliable micromethod for determination of the protein content in tissue homogenates. Acta Biol. Med. Ger. 1975;34:1441–1446
- Molecular determinants of altered Ca2+ handling in human chronic atrial fibrillation. Circulation. 2006;114:670–680
- Protein phosphatase activity is increased in a rat model of long-term beta-adrenergic stimulation. Naunyn- Schmiedeberg's Arch. Pharmacol. 2000;362:222–231
- L-type Ca2+ current downregulation in chronic human atrial fibrillation is associated with increased activity of protein phosphatases. Circulation. 2004;110:2651–2657
- . Ionic remodeling underlying action potential changes in a canine model of atrial fibrillation. Circ. Res. 1997;81:512–525
- . Ionic mechanisms of electrical remodeling in human atrial fibrillation. Cardiovasc. Res. 1999;44:121–131
- Effects of experimental heart failure on atrial cellular and ionic electrophysiology. Circulation. 2000;101:2631–2638
- Attrial amyloidosis: an arrhythmogenic substrate for persistent atrial fibrillation. Circulation. 2002;106:2091–2097
- Time course of atrial fibrillation-induced cellular structural remodeling in Atria of the goat. J. Mol. Cell. Cardiol. 2001;33:2083–2094
- . Excitation-Contraction Coupling and Cardiac Contractile Force 2nd edition Dordrecht. the Netherlands: Kluwer; 2001;
- . Altered cardiac myocyte Ca regulation in heart failure. Physiology (Bethesda). 2006;21:380–387
- . Protein kinase A-mediated acceleration of the stretch activation response in murine skinned myocardium is eliminated by ablation of cMyBP-C. Circ. Res. 2006;99:884–890
- Decreased phosphorylation levels of cardiac myosin-binding protein-C in human and experimental heart failure. J. Mol. Cell. Cardiol. 2007;43:223–229
- PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts. Cell. 2000;101:365–376
- Atrial fibrillation is associated with increased spontaneous calcium release from the sarcoplasmic reticulum in human atrial myocytes. Circulation. 2004;110:1358–1363
- . Phospholamban: a crucial regulator of cardiac contractility. Nat. Rev Mol. Cell Biol. 2003;4:566–577
- Defective cardiac ryanodine receptor regulation during atrial fibrillation. Circulation. 2005;111:2025–2032
- Calcium handling abnormalities underlying atrial arrhythmogenesis and contractile dysfunction in dogs with congestive heart failure. Circ. Arrhythmia Electrophysiol. 2008;1:93–102
- Myosin heavy chain composition and the economy of contraction in healthy and diseased human myocardium. J. Muscle Res. Cell Motil. 2005;26:39–48
- Alterations in contractile protein composition and function in human atrial dilatation and atrial fibrillation. J. Mol. Cell. Cardiol. 2006;41:467–477
PII: S0022-2828(08)01403-X
doi: 10.1016/j.yjmcc.2008.11.012
© 2008 Elsevier Inc. All rights reserved.
« Previous
Next »
Journal of Molecular and Cellular Cardiology
Volume 46, Issue 3
, Pages 385-394
, March 2009
